JONATHAN ROSAND, M.D., M.Sc.
Jonathan Rosand is Director of the Division of Neurocritical Care and Emergency Neurology, Medical Director of the Neuroscience Intensive Care Unit, and is an independent faculty member within the Center for Human Genetic Research at Massachusetts General Hospital. As a vascular and critical care neurologist with expertise in the genetics of complex disease, Dr. Rosands work has had a substantial impact through his roles as investigator, clinician, mentor and administrator. Motivated by his passion for patient care and commitment to rigorous science, Dr. Rosand ensures that his group embraces new ideas and technologies in a highly interdisciplinary environment.
Dr. Rosand is also a faculty member within the Program in Medical and Population Genetics of the Broad Institute. Prior to becoming the founding director of the Division of Neurocritical Care and Emergency Neurology, he directed the Massachusetts General Hospital/Brigham and Women's Hospital/Harvard Medical School Fellowship Training Program in Vascular and Critical Care Neurology. This program, the largest in the United States, trains neurologists in all aspects of stroke care and critical care neurology.
The Rosand lab has become a global leader in stroke genetics. Dr. Rosand established the International Stroke Genetics Consortium in 2007, and has served in a leadership capacity ever since. The ISGC has been responsible for identifying the majority of the currently established common genetic risk factors for stroke and has established a collaborative framework that has secured multi-center awards from the NIH, as well as awards from the Wellcome Trust in the UK and the Australian National Research Council. The Rosand lab's location within the CHGR and Broad has enabled it serve as scientific as well as the organizational leaders of the ISGC.
Projects in the lab continue to define role of genetic variation in cerebrovascular disease and recovery from brain injury, with the aim of ultimately translating discoveries to the bedside as rapidly as possible. In parallel, lab members continue to work closely with collaborators to refine the imaging of acute brain hemorrhage in order to identify biological pathways that can ultimately be exploited to improve recovery from these devastating injuries.